intracellular ATP, 
used to mimic increased extracellular glucose or reduced conductance 
of K(ATP) channels, enhanced the firing of action potentials in model 
neurons. The stochastic increases in intracellular ATP levels also 
demonstrated an irregular and clustered neuronal firing pattern. This 
phenomenon of K(ATP) channel attenuation could be one of the underlying 
mechanisms of glucose-related neuronal hyperexcitability and propagation.

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To run the models:
XPP: start with the command