 analyzed to investigate the role of K(ATP) channels in 
the firing of simulated action potentials. We found that incremental 
extracellular glucose could attenuate the activities of hippocampal 
K(ATP) channels. The effect was concentration dependent and involved 
mainly in open probabilities, not single-channel conductance. 
Additionally, higher levels of extracellular glucose could enhance 
neuropropagation; this could be attenuated by diazoxide, a K(ATP) 
channel agonist. In simulations, high levels of 