<reponame>hltfbk/E3C-Corpus
{
  "authors": [
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    },
    {
      "author": "<NAME>"
    }
  ],
  "doi": "10.4076/1752-1947-3-7225",
  "publication_date": "2009-10-16",
  "id": "EN118314",
  "url": "https://pubmed.ncbi.nlm.nih.gov/19830138",
  "source": "Journal of medical case reports",
  "source_url": "",
  "licence": "CC BY",
  "language": "en",
  "type": "pubmed",
  "description": "",
  "text": "Two patients, a 52-year-old man (Patient 1) and a 76-year-old man (Patient 2) with gliomatosis cerebri II were admitted to our institution; they underwent surgical treatment and received radiotherapy and chemotherapy. At the 24-month follow-up, Patient 1 was still alive, while Patient 2 had died. The poor prognosis of Patient 2 was underlined by molecular analysis which showed that the angiogenesis related genes VCAM1 and VEGF were overexpressed, reflecting the high degree of neovascularization."
}