This is an excellent idea. We have added an additional (final) main text figure, Fig. 7, illustrating how the molecular defects could converge on similar NMDA-EPSCs. We have explained the model in the accompanying figure legend with reference to supporting evidence in other figures.
Particularly, GluN2B GoF and LoF mutants show similar phenotypes in the presence of GluN1/2A/2B triheteromers, while the LoF mutation, C456Y, shows much less rescue effect than GoF mutation in the absence of both GluN2A and GluN2B.