If higher PGRMC1 levels are indeed critical for the suppression of decidualization, as is reasoned by the authors, then it should be possible to maintain PGRMC1 levels artificially to inhibit decidualization, as discussed below.
28) Lines 281-5. PGRMC1 (Hpr6.6) increased the rate of cell death (in a non-apoptotic mechanism) in MCF-7 cancer cells in response to H2O2 (PMID: 14523988). That could be discussed here in terms of mechanism.